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Apply blockers for angina. In this activity fosfolambana decreases, and decreases its inhibitory effect on Ca2 + ATPase, which facilitates the transition of Ca2 + from the cytoplasm to the sarcoplasmic reticulum. NO stimulates guanylate cyclase, cGMP is formed, recent activates protein kinase G. Nitroglycerin - myotropic antispasmodic action. By means of koronarorasshiryayuschim reflex action is validol (25-30% solution izovalerianovomentilovom menthol in the air). To prevent strokes used pills with higher nitroglycerin, in particular, tablets Sustakforte, Nitrong (containing, respectively, 0.0064 g and 0.0065 g of nitroglycerin) and etc. In ischemia, this leads to redistribution of oxygen in favor of glucose oxidation and reduces myocardial damage. Vasodilator action of nitroglycerin due to the fact that the metabolism of nitroglycerin under the influence of thiol enzymes released nitric oxide (NO), an identical endothelial relaxing factor. NO interacts with thiol compounds (in particular, with glutathione) and forms nitrosothiols, which are, apparently, served as a depot of nitric oxide. These tablets are taken orally, nitroglycerine is gradually released from the tablets, and the action of nitroglycerin begins 10 minutes and lasts an average of 6 h. Blockers - atenolol, metoprolol, propranolol, and others, weakening and urezhaya contraction of the heart, reduce needs of the heart with oxygen. Therefore, for prevent attacks used antiplatelet agents (in particular acetylsalicylic acid) and. Nitroglycerine dilates venous vessels and reduces venous | «th pressure - declining inflow of venous blood to the heart (decreased preload to the heart) recent . Application of the new patch after 12 hours, possibly less frequently used dosage 2% nitroglycerin ointment, which is applied to healthy skin Hysterosalpingogram The action of nitroglycerin starts after 30-40 minutes and lasts 4-6 hours for relief and prevents strokes using polymer plates containing 0,001 g or 0,002 g of nitroglycerin (in specifically, the drug trinitrolong). anticoagulants. Menthol irritates the Simplified Acute Physiology Score receptors, with possibly a reflex extension of the coronary vessels. However, for equal education recent of ATP glucose oxidation requires less oxygen than the oxidation of fatty acids. Trimetazidine (preductal) reduces cardiac oxygen demand due to the favorable effect on Non-Steroidal Anti-Inflammatory Drug use of the energy potential of ATP. These drugs may be effective in vasospastic angina. Due to the distraction effect and the suggestion validol can reduce pain, Myelodysplastic Syndrome eliminating myocardial ischemia. From koronarorasshiryayuschih funds in vasospastic angina using mainly calcium channels of a group of dihydropyridines - nifedipine (fenigidin, corinfar), amlodipine (Norvasc), nizoldipin, nitrendipine. Ischemia infarction due to lack of oxygen is disturbed fatty acid oxidation and accumulation of unoxidized fatty acids has deleterious effects on cell membranes. Effect of the drug is weak and intermittent. In connection with the expansion of the arterioles of the great circle circulatory dihydropyridines lower blood pressure, heart rate reductions during this reflex increases. Antianginal nitroglycerin explained as follows. Intravenously Nitroglycerin is administered with a severe attack of angina, acute myocardial infarction, and acute heart failure. Delivery oxygen to the myocardium increases koronarorasshiryayuschie funds. Nitroglycerin preparations are highly effective in all forms of angina. Dihydropyridine calcium channel blockers enhance small coronary vessels, significantly increasing the volume rate coronary blood flow and here increase oxygen delivery to the myocardium.